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  Indian J Med Microbiol
 

Figure 1 Photograph of transverse sections of the aorta. (Slide 1-I): Transverse section of the aorta of control groups (Groups Ia, IIa & IIIa) showing normal histological structures of intima, media and adventitia. (Slide 1-Ib): Transverse section of the aorta of group Ib shows focal areas of intimal thickening (A) with atheromatous cap formation and intimal protrusion into lumen (B). (Slide1- IIb): Transverse section of the aorta of group IIb shows focal areas of intimal thickening (A) and atheromatous cap formation with intimal protrusion into lumen (B) with focal ulceration of intimal lining (C) with focal atrophy of media (D). (Slide1- IIIb): Transverse section of the aorta of group IIIb shows focal areas of intimal thickening with sub-intimal proliferation and migration of smooth muscle cells associated with extra cellular matrix deposition (A) and intimal protrusion into lumen (B) with focal ulceration; by higher power (slide 1-IIIbH): there is intimal proliferation and subintimal migration of smooth muscle cells, macrophages and mononuclear cells infiltration, formation of foam cells with extracellular needle-like crystal of fat. (Slides1- Ic, IIc & IIIc): Transverse section of the aorta of Groups Ic, IIc & IIIc show The thickness of the walls of the aortas and histological structure of these groups similar to those of the control groups except presence of intimal protrusion into lumen (B) in all treated groups and remnants of intimal damage in the form of residual ulceration (C) in group IIIC (slid1- IIIC).

Figure 1 Photograph of transverse sections of the aorta. (Slide 1-I): Transverse section of the aorta of control groups (Groups Ia, IIa & IIIa) showing normal histological structures of intima, media and adventitia. (Slide 1-Ib): Transverse section of the aorta of group Ib shows focal areas of intimal thickening (A) with atheromatous cap formation and intimal protrusion into lumen (B). (Slide1- IIb): Transverse section of the aorta of group IIb shows focal areas of intimal thickening (A) and atheromatous cap formation with intimal protrusion into lumen (B) with focal ulceration of intimal lining (C) with focal atrophy of media (D). (Slide1- IIIb): Transverse section of the aorta of group IIIb shows focal areas of intimal thickening with sub-intimal proliferation and migration of smooth muscle cells associated with extra cellular matrix deposition (A) and intimal protrusion into lumen (B) with focal ulceration; by higher power (slide 1-IIIbH): there is intimal proliferation and subintimal migration of smooth muscle cells, macrophages and mononuclear cells infiltration, formation of foam cells with extracellular needle-like crystal of fat. (Slides1- Ic, IIc & IIIc): Transverse section of the aorta of Groups Ic, IIc & IIIc show The thickness of the walls of the aortas and histological structure of these groups similar to those of the control groups except presence of intimal protrusion into lumen (B) in all treated groups and remnants of intimal damage in the form of residual ulceration (C) in group IIIC (slid1- IIIC).